Search results for "Endothelin A Receptor Antagonists"

showing 6 items of 6 documents

Anti-Hypotensive Treatment and Endothelin Blockade Synergistically Antagonize Exercise Fatigue in Rats under Simulated High Altitude

2013

Rapid ascent to high altitude causes illness and fatigue, and there is a demand for effective acute treatments to alleviate such effects. We hypothesized that increased oxygen delivery to the tissue using a combination of a hypertensive agent and an endothelin receptor A antagonist drugs would limit exercise-induced fatigue at simulated high altitude. Our data showed that the combination of 0.1 mg/kg ambrisentan with either 20 mg/kg ephedrine or 10 mg/kg methylphenidate significantly improved exercise duration in rats at simulated altitude of 4,267 m, whereas the individual compounds did not. In normoxic, anesthetized rats, ephedrine alone and in combination with ambrisentan increased heart…

Critical Care and Emergency MedicinePulmonologyPhysiologyAcclimatizationRespiratory Systemlcsh:MedicineAltitude SicknessPharmacologyCardiovascular PhysiologyDrug DiscoveryMedicine and Health SciencesDrug InteractionsSympathomimeticslcsh:ScienceFatigueAltitude sicknessEphedrineMammalsMultidisciplinaryPhenylpropionatesAltitudeDrug SynergismHematologyAnimal ModelsCell HypoxiaPyridazinesmedicine.anatomical_structureVertebratesBlood CirculationDrug Therapy CombinationAnatomymedicine.symptomEndothelin receptorPerfusionInjections IntraperitonealResearch Articlemedicine.drugDrug Research and DevelopmentAmbrisentanEndothelin A Receptor AntagonistsCardiologyEnvironmental and Occupational Lung DiseasesResearch and Analysis MethodsRodentsCardiovascular PharmacologyModel OrganismsHeart ratemedicineAnimalsRespiratory PhysiologySports and Exercise MedicinePharmacologyDose-Response Relationship Drugbusiness.industryAcute Cardiovascular Problemslcsh:ROrganismsHemodynamicsBiology and Life SciencesSkeletal muscleHypoxia (medical)medicine.diseaseRatsDisease Models AnimalBlood pressureMethylphenidateCardiovascular Anatomylcsh:QClinical MedicinebusinessPLoS ONE
researchProduct

Inflammatory Murine Skin Responses to UV-B Light Are Partially Dependent on Endothelin-1 and Mast Cells

2006

Endothelin (ET-1) has been shown to crucially contribute to UV-induced skin responses such as tanning. To test whether ET-1 is also involved in early cutaneous reactions to UV, we assessed ET-1 skin levels in UV-irradiated mice. In correlation with the levels of UV-induced skin inflammation, ET-1 concentrations increased substantially and continually. Moreover, blocking of ET-1 receptors (ET A ) resulted in significantly decreased cutaneous inflammation following UV irradiation. When we assessed skin responses to ET-1 injections, we observed prominent mast cell degranulation and mast cell-dependent inflammation. Since mast cells also critically contributed to UV-induced inflammation, we det…

Endothelin A Receptor AntagonistsUltraviolet RaysCell DegranulationRatónDermatitisMice TransgenicInflammationBiologyPharmacologyCell DegranulationPathology and Forensic MedicineMicemedicineAnimalsMast CellsReceptorInflammationEndothelin-1integumentary systemDegranulationDose-Response Relationship RadiationReceptor Endothelin AMast cellEndothelin 1medicine.anatomical_structureImmunologymedicine.symptomEndothelin receptorRegular ArticlesThe American Journal of Pathology
researchProduct

Mechanisms underlying diabetes enhancement of endothelin-1-induced contraction in rabbit basilar artery

2004

The influence of alloxan-induced diabetes on the reactivity of rabbit basilar artery to endothelin-1 was examined. Endothelin-1 induced concentration-dependent contraction of basilar arteries that was higher in diabetic than in control rabbits. Endothelium removal produced a higher enhancement of the endothelin-1-induced contraction in control than in diabetic rabbits. N(G)-nitro-L-arginine (L-NOArg) enhanced the maximal contraction induced by endothelin-1 in control rabbits and potentiated this response in diabetic rabbits. Endothelin ETA receptor antagonist, cyclo(D-Asp-Pro-D-Val-Leu-D-Trp) (BQ-123), inhibited endothelin-1-induced contraction in both rabbit groups. Endothelin ETB receptor…

Endothelin Receptor AntagonistsMaleNitroprussidemedicine.medical_specialtyContraction (grammar)Vascular smooth muscleEndotheliumEndothelin A Receptor AntagonistsVasodilator AgentsEndothelin B Receptor AntagonistsNitroargininePeptides CyclicMuscle Smooth VascularDiabetes Mellitus ExperimentalPiperidinesIsometric Contractionmedicine.arteryInternal medicinemedicineBasilar arteryAnimalsEnzyme InhibitorsAntihypertensive AgentsPharmacologyDiabetisEndothelin-1Artèriesbusiness.industryEndoteli vascularReceptor Endothelin AReceptor Endothelin BEndothelin 1Òxid nítricEndothelin A Receptor AntagonistsEndothelin B Receptor AntagonistsEndocrinologymedicine.anatomical_structureBasilar Arterycardiovascular systemRabbitsbusinessEndothelin receptorOligopeptidesEuropean Journal of Pharmacology
researchProduct

Different role of endothelin ETA and ETB receptors and endothelial modulators in diabetes-induced hyperreactivity of the rabbit carotid artery to end…

2004

The influence of diabetes on regulatory mechanisms and specific receptors implicated in the contractile response of isolated rabbit carotid arteries to endothelin-1 was examined. Endothelin-1 induced a concentration-dependent contraction that was greater in arteries from diabetic rabbits than in arteries from control rabbits. Endothelium removal or N(G)-nitro-L-arginine enhanced contractions in response to endothelin-1 only in control arteries, without modifying the endothelin-1 response in diabetic arteries. Indomethacin, furegrelate (thromboxane A(2) inhibitor), or cyclo-(D-Asp-Pro-D-Val-Leu-D-Trp) (BQ-123; endothelin ET(A) receptor antagonist) inhibited the contractions in response to en…

Malemedicine.medical_specialtyEndotheliumCarotid Artery CommonEndothelin A Receptor AntagonistsThromboxanemedicine.drug_classEndothelin B Receptor AntagonistsIn Vitro TechniquesDiabetes Mellitus ExperimentalInternal medicinemedicineAnimalsReceptorPharmacologyDose-Response Relationship DrugEndothelin-1business.industryReceptor Endothelin AReceptor antagonistReceptor Endothelin BEndothelin 1Endothelin A Receptor AntagonistsEndothelin B Receptor Antagonistsmedicine.anatomical_structureEndocrinologyVasoconstrictioncardiovascular systemEndothelium VascularRabbitsEndothelin receptorbusinessEuropean Journal of Pharmacology
researchProduct

Endothelin receptor expression in idiopathic pulmonary arterial hypertension: effect of bosentan and epoprostenol treatment.

2011

Endothelin receptor antagonists are used to treat idiopathic pulmonary arterial hypertension (IPAH), but human pulmonary arterial endothelin receptor expression is not well defined. We hypothesised that disease and treatment would modify normal receptor distribution in pulmonary resistance arteries of children. Using immunohistochemistry and semiquantitative analysis, we investigated endothelin receptor subtypes A and B (ET(A) and ET(B), respectively), and endothelial nitric oxide synthase (eNOS) expression in peripheral pulmonary arteries of tissue from untreated children with IPAH (n=7), following extended combined bosentan and epoprostenol therapy (n=5) and from normal subjects (n=5). Cl…

Pulmonary and Respiratory MedicineMalePathologymedicine.medical_specialtyEndotheliumAdolescentNifedipineNitric Oxide Synthase Type IIIEndothelin A Receptor AntagonistsEndothelin B Receptor AntagonistsHypertension PulmonaryVasodilator AgentsPulmonary ArteryMuscle Smooth VascularPiperazinesSildenafil CitrateNitric oxidechemistry.chemical_compoundEnosInternal medicinemedicineHumansFamilial Primary Pulmonary HypertensionSulfonesReceptorChildAntihypertensive AgentsSulfonamidesbiologybusiness.industryBosentanbiology.organism_classificationReceptor Endothelin AEpoprostenolReceptor Endothelin BEndothelin A Receptor AntagonistsBosentanEndothelin B Receptor Antagonistsmedicine.anatomical_structureEndocrinologychemistryPurinesChild PreschoolDrug Therapy CombinationFemaleEndothelium VascularEndothelin receptorbusinessmedicine.drugThe European respiratory journal
researchProduct

EndothelinA receptor antagonist BSF-208075 causes immune modulation and neuroprotection after stroke in gerbils.

2005

Leukocytes contribute to the ischemia-reperfusion injury. Recent studies suggested endothelins could be important mediators for leukocyte activation in stroke. We tested if the endothelinA receptor antagonist BSF-208075 (ambrisentan) could reduce an ischemic lesion by modulation of leukocyte-endothelium interactions. Twenty-four gerbils underwent either a sham operation (n=6) or 15 min of bilateral carotid artery occlusion resulting in global cerebral ischemia. Ischemic animals received normal saline (n=6), 5 mg/kg BSF-208075 (n=6) or 30 mg/kg (n=6) administered intravenously at 10 min of reperfusion. Leukocytes rolling or adhering to endothelium were counted by intravital microscopy in par…

medicine.hormoneMaleAmbrisentanmedicine.drug_classEndothelin A Receptor AntagonistsIschemiaPharmacologyBrain IschemiaEndothelinsLeukocytesMedicineAnimalsImmunologic FactorsMolecular BiologyStrokeDose-Response Relationship DrugPhenylpropionatesbusiness.industryGeneral NeuroscienceEndothelinsAntagonistBrainmedicine.diseaseReceptor antagonistReceptor Endothelin APyridazinesStrokeChemotaxis LeukocyteNeuroprotective AgentsTreatment OutcomeAnesthesiaCerebrovascular CirculationReperfusion InjuryNerve DegenerationEncephalitisNeurology (clinical)businessEndothelin receptorGerbillinaeIntravital microscopyDevelopmental Biologymedicine.drugBrain research
researchProduct